Preliminary experiments show conolidine may possibly inhibit particular ion channels, minimizing neuronal excitability and limiting discomfort alerts. This mechanism is particularly suitable in neuropathic soreness, wherever irregular signaling will cause persistent discomfort. Additionally, conolidine appears to influence G protein-coupled receptor (GPCR) pathways integral to agony n... https://deborahs332lvf4.wikicommunication.com/user