Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Alternatively, our power to rescue SERT operate in KI midbrain synaptoneurosomes via the inhibition of FAK implies elevated FAK signaling downstream with the Pro32Pro33 mutant, as confirmed by improved pFAK localization in 5-HT synapses. Our https://patrickh034cvo8.loginblogin.com/profile