Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Then again, our capability to rescue SERT perform in KI midbrain synaptoneurosomes through the inhibition of FAK indicates elevated FAK signaling downstream of the Pro32Pro33 mutant, as verified by enhanced pFAK localization in 5-HT synapses. https://rafaeljvgqb.angelinsblog.com/31547416/fascination-about-pro33